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Pulp necrosis following luxated injury to teeth in a patient with uncontrolled type II diabetes mellitus: a case report
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Case Report Pulp necrosis following luxated injury to teeth in a patient with uncontrolled type II diabetes mellitus: a case report
Haneol Shin, DDS, Seung-Jong Lee, DDS, MS, Il-Young Jung, DDS, MSD, PhD, Chan-Young Lee, DDS, MSD, DDSc
Restorative Dentistry & Endodontics 2012;37(1):61-65.
DOI: https://doi.org/10.5395/rde.2012.37.1.61
Published online: March 2, 2012

Department of Conservative Dentistry, Yonsei University College of Dentistry, Seoul, Korea.

Correspondence to Chan-Young Lee, DDS, MSD, DDSc. Professor, Department of Conservative Dentistry, Yonsei University College of Dentistry, 50 Yonsei-ro, Seodaemun-gu, Seoul, Korea 120-752. TEL, +82-2-2228-8701; FAX, +82-2-313-7575; chanyoungl@yuhs.ac
• Received: December 16, 2011   • Revised: January 16, 2012   • Accepted: January 20, 2012

©Copyights 2012. The Korean Academy of Conservative Dentistry.

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  • Patients with diabetes mellitus show delayed wound healing and increased susceptibility to infection. Therefore, the effects of diabetes on pulpal and periodontal healing should be taken into consideration when treating diabetic dental traumatized patients. This case presents the treatment for dental traumatized 20 yr old female with uncontrolled type II diabetes. The traumatized upper central incisors had showed pulpal healing in early days. However, 7 mon after the trauma, the teeth had been diagnosed with pulp necrosis with apical abscess. Eventually, non surgical root canal treatment on the teeth had been performed.
  • Keywords: Diabetes mellitus; Luxation; Pulp necrosis; Transient apical breakdown
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Figure 1
Radiograph of maxillary central incisors after traumatic injury.
rde-37-61-g001.jpg
Figure 2
(a) Radiograph was taken 2 weeks after traumatic injury. Small periapical radiolucencies associated both central incisors were observed; (b) Radiograph was taken 6 weeks after traumatic injury. Root resorptions as well as periapical radiolucencies in both teeth were evident; (c) Radiograph was taken 6 months after traumatic injury. There were no definite changes in root resorptions and periapical radiolucencies compared to radiograph at 6 weeks after trauma; (d) Radiograph was taken 7 months after traumatic injury. Periapical radiolucency of upper left central incisal had been increased; (e) Radiograph was taken after endodontic treatment on maxillary left central incisor. Note the increase of periapical radiolucency on right central incisor.
rde-37-61-g002.jpg
Figure 3
(a) Ultrasound Doppler images of upper anterior teeth at 6 weeks after traumatic injury. Pulsating signs in both upper central incisors were not clear like upper left lateral incisor, but observed faintly; (b) Ultrasound Doppler image of upper anterior teeth at 6 months after traumatic injury. Distinct pulsating sign were observed in all 4 anterior teeth; (c) Ultrasound Doppler images of upper anterior teeth at 7 months after traumatic injury. Pulsating sign on upper left central incisor had been disappeared; (d) Ultrasound Doppler images of upper anterior teeth at 11 months after traumatic injury. Pulsating sign on upper right central incisor was disappeared. Left central incisor had been root canal treated (negative control) and had no pulsating sign either.
rde-37-61-g003.jpg
Figure 4
Radiograph was taken after endodontic treatment on right upper central incisor. Periapical radiolucencies on both teeth were remained.
rde-37-61-g004.jpg

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        Pulp necrosis following luxated injury to teeth in a patient with uncontrolled type II diabetes mellitus: a case report
        Restor Dent Endod. 2012;37(1):61-65.   Published online March 2, 2012
        DOI: https://doi.org/10.5395/rde.2012.37.1.61
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      Pulp necrosis following luxated injury to teeth in a patient with uncontrolled type II diabetes mellitus: a case report
      Image Image Image Image
      Figure 1 Radiograph of maxillary central incisors after traumatic injury.
      Figure 2 (a) Radiograph was taken 2 weeks after traumatic injury. Small periapical radiolucencies associated both central incisors were observed; (b) Radiograph was taken 6 weeks after traumatic injury. Root resorptions as well as periapical radiolucencies in both teeth were evident; (c) Radiograph was taken 6 months after traumatic injury. There were no definite changes in root resorptions and periapical radiolucencies compared to radiograph at 6 weeks after trauma; (d) Radiograph was taken 7 months after traumatic injury. Periapical radiolucency of upper left central incisal had been increased; (e) Radiograph was taken after endodontic treatment on maxillary left central incisor. Note the increase of periapical radiolucency on right central incisor.
      Figure 3 (a) Ultrasound Doppler images of upper anterior teeth at 6 weeks after traumatic injury. Pulsating signs in both upper central incisors were not clear like upper left lateral incisor, but observed faintly; (b) Ultrasound Doppler image of upper anterior teeth at 6 months after traumatic injury. Distinct pulsating sign were observed in all 4 anterior teeth; (c) Ultrasound Doppler images of upper anterior teeth at 7 months after traumatic injury. Pulsating sign on upper left central incisor had been disappeared; (d) Ultrasound Doppler images of upper anterior teeth at 11 months after traumatic injury. Pulsating sign on upper right central incisor was disappeared. Left central incisor had been root canal treated (negative control) and had no pulsating sign either.
      Figure 4 Radiograph was taken after endodontic treatment on right upper central incisor. Periapical radiolucencies on both teeth were remained.

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      Pulp necrosis following luxated injury to teeth in a patient with uncontrolled type II diabetes mellitus: a case report
      Restor Dent Endod : Restorative Dentistry & Endodontics
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