Warning: mkdir(): Permission denied in /home/virtual/lib/view_data.php on line 81

Warning: fopen(upload/ip_log/ip_log_2024-12.txt): failed to open stream: No such file or directory in /home/virtual/lib/view_data.php on line 83

Warning: fwrite() expects parameter 1 to be resource, boolean given in /home/virtual/lib/view_data.php on line 84
Vital tooth with periapical lesion: spontaneous healing after conservative treatment

Vital tooth with periapical lesion: spontaneous healing after conservative treatment

Article information

Restor Dent Endod. 2012;37(2):123-126
Publication date (electronic) : 2012 May 18
doi : https://doi.org/10.5395/rde.2012.37.2.123
Department of Conservative Dentistry, Yonsei University College of Dentistry, Seoul, Korea.
Correspondence to Sung-Ho Park, DDS, PhD. Professor, Department of Conservative Dentistry, Yonsei University College of Dentistry, 134 Shinchon-dong, Seodaemun-gu, Seoul, Korea 120-752. TEL, +82-2-2228-3147; FAX, +82-2-313-7575; sunghopark@yuhs.ac
Received 2011 December 27; Revised 2012 March 05; Accepted 2012 April 06.

Abstract

It is often presumed that apical periodontitis follows total pulp necrosis, and consequently root canal treatment is commonly performed. Periapical lesion development is usually caused by bacteria and its byproduct which irritate pulp, develop pulpitis, and result in necrosis through an irreversible process. Afterwards, apical periodontitis occurs. This phenomenon is observed as an apical radiolucency in radiographic view. However, this unusual case presents a spontaneous healing of periapical lesion, which has developed without pulp necrosis in a vital tooth, through conservative treatment.

Notes

No potential conflict of interest relevant to this article was reported.

References

1. Kim IS, Kim SG, Kim YK, Kim JD. Position of the mental foramen in a Korean population: a clinical and radiographic study. Implant Dent 2006. 15404–411.
2. Resnick CM, Novelline RA. Cemento-osseous dysplasia, a radiological mimic of periapical dental abscess. Emerg Radiol 2008. 15367–374.
3. Romanos GE, Froum S, Costa-Martins S, Meitner S, Tarnow DP. Implant periapical lesions: etiology and treatment options. J Oral Implantol 2011. 3753–63.
4. Kovacević M, Tamarut T, Jonjić N, Braut A. The transition from pulpitis to periapical periodontitis in dogs' teeth. Aust Endod J 2008. 3412–18.
5. Khayat BG, Byers MR, Taylor PE, Mecifi K, Kimberly CL. Responses of nerve fibers to pulpal inflammation and periapical lesions in rat molars demonstrated by calcitonin gene-related peptide immunocytochemistry. J Endod 1988. 14577–587.
6. Byers MR, Taylor PE, Khayat BG, Kimberly CL. Effects of injury and inflammation on pulpal and periapical nerves. J Endod 1990. 1678–84.
7. Caviedes-Bucheli J, Muñoz HR, Azuero-Holguín MM, Ulate E. Neuropeptides in dental pulp: the silent protagonists. J Endod 2008. 34773–788.
8. Stashenko P, Teles R, D'Souza R. Periapical inflammatory responses and their modulation. Crit Rev Oral Biol Med 1998. 9498–521.

Article information Continued

Figure 1

Preoperative periapical view and clinical photograph. (a) Periapical radiolucency on #35; (b) Cervical abrasion.

Figure 2

Preoperative panorama view. Periapical radiolucency was observed at the apex of mandibular left second premolar.

Figure 3

Periapical view taken 2 years ago.

Figure 4

Location of mental foramens (dotted line) and periapical radiolucency (arrow).

Figure 5

Periapical view and clinical photograph after class V Resin filling.

Figure 6

Three-month follow up. (a) Periapical view. Distal proximal caries was detected; (b) Photograph taken after class II resin filling.

Figure 7

Six-month follow up. Periapical view.

Figure 8

Nine-month follow up. Periapical view.

Figure 9

Fourteen-month follow up. Clinical photographs.

Figure 10

Fourteen-month follow up. Horizontal shift periapical view.

Figure 11

One year before implant surgery: Note that the posterior area of mandibular left segment was edentulous.